H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor | |
Libo Su; Wenlong Xia; Tianjin Shen; Qingli Liang; Wenwen Wang; Li H(李红); Jiao JW(焦建伟) | |
刊名 | NUCLEIC ACIDS RESEARCH
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2018 | |
卷号 | 46期号:17页码:8817-8831 |
内容类型 | 期刊论文 |
源URL | [http://ir.ioz.ac.cn/handle/000000/13400] ![]() |
专题 | 干细胞与生殖生物学国家重点实验室 |
推荐引用方式 GB/T 7714 | Libo Su,Wenlong Xia,Tianjin Shen,et al. H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor[J]. NUCLEIC ACIDS RESEARCH,2018,46(17):8817-8831. |
APA | Libo Su.,Wenlong Xia.,Tianjin Shen.,Qingli Liang.,Wenwen Wang.,...&焦建伟.(2018).H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor.NUCLEIC ACIDS RESEARCH,46(17),8817-8831. |
MLA | Libo Su,et al."H2A.Z.1 crosstalk with H3K56-acetylation controls gliogenesis through the transcription of folate receptor".NUCLEIC ACIDS RESEARCH 46.17(2018):8817-8831. |
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