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IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice.
Zhang, Lisha; Liu, Peining; Wen, Wen; Bai, Xiaofang; Zhang, Yan; Liu, Mengping; Wang, Lijun; Wu, Yue; Yuan, Zuyi; Zhou, Juan
刊名Molecular Immunology
2019
卷号Vol.105页码:240-250
关键词CORONARY heart disease *LABORATORY mice *MACROPHAGES *INFLAMMASOMES *HEART diseases
ISSN号0161-5890
URL标识查看原文
公开日期[db:dc_date_available]
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/4740082
专题湖南大学
作者单位1.Department of Cardiovascular Medicine, The First Affiliated Hospital, Xi'an Jiaotong University, Xi'an, Shaanxi, PR China
2.Key Laboratory of Environment and Genes Related to Diseases , Ministry of Education, Xi'an, Shaanxi, PR China
3.Key Laboratory of Molecular Cardiology, Shaanxi Province, Xi'an, Shaanxi, PR China
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GB/T 7714
Zhang, Lisha,Liu, Peining,Wen, Wen,et al. IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice.[J]. Molecular Immunology,2019,Vol.105:240-250.
APA Zhang, Lisha.,Liu, Peining.,Wen, Wen.,Bai, Xiaofang.,Zhang, Yan.,...&Zhou, Juan.(2019).IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice..Molecular Immunology,Vol.105,240-250.
MLA Zhang, Lisha,et al."IL-17A contributes to myocardial ischemic injury by activating NLRP3 inflammasome in macrophages through AMPKα/p38MAPK/ERK1/2 signal pathway in mice.".Molecular Immunology Vol.105(2019):240-250.
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