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	RAS-MAPK Reactivation Facilitates Acquired Resistance in FGFR1-Amplified Lung Cancer and Underlies a Rationale for Upfront FGFR-MEK Blockade
	Rusan, Maria; Bockorny, Bruno; Chen, Wankun; Liao, Rachel G.; Li, Yvonne; Piccioni, Federica; Wang, Jun; Tan, Li; Thorner, Aaron R.; Li, Tianxia
刊名	MOLECULAR CANCER THERAPEUTICS
	2018
卷号	17期号:7
ISSN号	1535-7163
URL标识	查看原文
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3599821
专题	复旦大学上海医学院
推荐引用方式 GB/T 7714	Rusan, Maria,Bockorny, Bruno,Chen, Wankun,et al. RAS-MAPK Reactivation Facilitates Acquired Resistance in FGFR1-Amplified Lung Cancer and Underlies a Rationale for Upfront FGFR-MEK Blockade[J]. MOLECULAR CANCER THERAPEUTICS,2018,17(7).
APA	Rusan, Maria.,Bockorny, Bruno.,Chen, Wankun.,Liao, Rachel G..,Li, Yvonne.,...&Bass, Adam J..(2018).RAS-MAPK Reactivation Facilitates Acquired Resistance in FGFR1-Amplified Lung Cancer and Underlies a Rationale for Upfront FGFR-MEK Blockade.MOLECULAR CANCER THERAPEUTICS,17(7).
MLA	Rusan, Maria,et al."RAS-MAPK Reactivation Facilitates Acquired Resistance in FGFR1-Amplified Lung Cancer and Underlies a Rationale for Upfront FGFR-MEK Blockade".MOLECULAR CANCER THERAPEUTICS 17.7(2018).

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