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	Knockdown of Receptor Interacting Protein kinase-1 (RIPK1) markedly exacerbates immune-mediated liver injury and induces lethality through massive TNF alpha/caspase-dependent apoptosis of hepatocytes, independently of RIPK3/MLKL-mediated necroptosis.
	Suda, Jo; Dara, Lily; Yang, Luoluo; Gaarde, William A.; Kaplowitz, Neil; Liu, Zhang-Xu
刊名	HEPATOLOGY
	2015
卷号	62
ISSN号	0270-9139
URL标识	查看原文
语种	英语
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/3543284
专题	吉林大学白求恩第一医院
推荐引用方式 GB/T 7714	Suda, Jo,Dara, Lily,Yang, Luoluo,et al. Knockdown of Receptor Interacting Protein kinase-1 (RIPK1) markedly exacerbates immune-mediated liver injury and induces lethality through massive TNF alpha/caspase-dependent apoptosis of hepatocytes, independently of RIPK3/MLKL-mediated necroptosis.[J]. HEPATOLOGY,2015,62.
APA	Suda, Jo,Dara, Lily,Yang, Luoluo,Gaarde, William A.,Kaplowitz, Neil,&Liu, Zhang-Xu.(2015).Knockdown of Receptor Interacting Protein kinase-1 (RIPK1) markedly exacerbates immune-mediated liver injury and induces lethality through massive TNF alpha/caspase-dependent apoptosis of hepatocytes, independently of RIPK3/MLKL-mediated necroptosis..HEPATOLOGY,62.
MLA	Suda, Jo,et al."Knockdown of Receptor Interacting Protein kinase-1 (RIPK1) markedly exacerbates immune-mediated liver injury and induces lethality through massive TNF alpha/caspase-dependent apoptosis of hepatocytes, independently of RIPK3/MLKL-mediated necroptosis.".HEPATOLOGY 62(2015).

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