QQ客服 官方微博 反馈留言

	Novel ASK1 inhibitor AGI-1067 improves AGE-induced cardiac dysfunction by inhibiting MKKs/p38 MAPK and NF-kappa B apoptotic signaling
	Liu, Zhongwei; Zheng, Shixiang; Wang, Xi; Qiu, Chuan; Guo, Yan
刊名	FEBS OPEN BIO
	2018
卷号	8页码:1445-1456
关键词	advanced glycation end products apoptosis heart failure apoptosis signal-regulating kinase diabetes
ISSN号	2211-5463
URL标识	查看原文
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/2923035
专题	西安交通大学
推荐引用方式 GB/T 7714	Liu, Zhongwei,Zheng, Shixiang,Wang, Xi,et al. Novel ASK1 inhibitor AGI-1067 improves AGE-induced cardiac dysfunction by inhibiting MKKs/p38 MAPK and NF-kappa B apoptotic signaling[J]. FEBS OPEN BIO,2018,8:1445-1456.
APA	Liu, Zhongwei,Zheng, Shixiang,Wang, Xi,Qiu, Chuan,&Guo, Yan.(2018).Novel ASK1 inhibitor AGI-1067 improves AGE-induced cardiac dysfunction by inhibiting MKKs/p38 MAPK and NF-kappa B apoptotic signaling.FEBS OPEN BIO,8,1445-1456.
MLA	Liu, Zhongwei,et al."Novel ASK1 inhibitor AGI-1067 improves AGE-induced cardiac dysfunction by inhibiting MKKs/p38 MAPK and NF-kappa B apoptotic signaling".FEBS OPEN BIO 8(2018):1445-1456.

个性服务

查看访问统计

相关权益政策

暂无数据

收藏/分享

除非特别说明，本系统中所有内容都受版权保护，并保留所有权利。