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Niacin receptor GPR109A inhibits insulin secretion and is down-regulated in type 2 diabetic islet beta-cells
Wang, Na[1]; Guo, De-yu[4]; Tian, Xiong[1]; Lin, Hao-peng[1]; Li, Yun-pan[1]; Chen, Shao-jun[1]; Fu, Yu-cai[2]; Xu, Wen-can[3]; Wei, Chi-ju[1]
刊名GENERAL AND COMPARATIVE ENDOCRINOLOGY
2016
卷号237页码:98-108
关键词GPR109A Insulin secretion Niacin Diabetes Insulinoma
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/2195643
专题华南理工大学
作者单位1.[1]Shantou Univ, Multidisciplinary Res Ctr, Shantou 515063, Guangdong, Peoples R China
2.[2]Shantou Univ Med Coll, Lab Cell Senescence, Shantou 515041, Guangdong, Peoples R China
3.[3]Shantou Univ, Coll Med, Affiliated Hosp 1, Dept Endocrinol, Shantou 515041, Guangdong, Peoples R China
4.[4]Xinan Hosp Chongqing, Dept Pathol, Chongqing 400038, Peoples R China
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GB/T 7714
Wang, Na[1],Guo, De-yu[4],Tian, Xiong[1],et al. Niacin receptor GPR109A inhibits insulin secretion and is down-regulated in type 2 diabetic islet beta-cells[J]. GENERAL AND COMPARATIVE ENDOCRINOLOGY,2016,237:98-108.
APA Wang, Na[1].,Guo, De-yu[4].,Tian, Xiong[1].,Lin, Hao-peng[1].,Li, Yun-pan[1].,...&Wei, Chi-ju[1].(2016).Niacin receptor GPR109A inhibits insulin secretion and is down-regulated in type 2 diabetic islet beta-cells.GENERAL AND COMPARATIVE ENDOCRINOLOGY,237,98-108.
MLA Wang, Na[1],et al."Niacin receptor GPR109A inhibits insulin secretion and is down-regulated in type 2 diabetic islet beta-cells".GENERAL AND COMPARATIVE ENDOCRINOLOGY 237(2016):98-108.
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