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Melatonin decreases M1 polarization via attenuating mitochondrial oxidative damage depending on UCP2 pathway in prorenin-treated microglia
Hu, Li[1]; Zhang, Shutian[2]; Wen, Haoyu[3]; Liu, Tianfeng[4]; Cai, Jian[5]; Du, Dongshu[6]; Zhu, Danian[7]; Chen, Fuxue[8]; Xia, Chunmei[9]
刊名PLOS ONE
2019
卷号14页码:e0212138
ISSN号1932-6203
URL标识查看原文
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/2164206
专题上海大学
作者单位1.[1]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Shanghai Key Lab Bioenergy Crops, Coll Life Sci, Shanghai, Peoples R China.
2.[2]Fudan Univ, Dept Physiol & Pathophysiol, Basic Med Coll, Shanghai, Peoples R China.
3.[3]Fudan Univ, Dept Physiol & Pathophysiol, Basic Med Coll, Shanghai, Peoples R China.
4.[4]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Shanghai Key Lab Bioenergy Crops, Coll Life Sci, Shanghai, Peoples R China.
5.[5]Shanghai Jiao Tong Univ, Dept Neurol, Renji Hosp, Shanghai, Peoples R China.
6.[6]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Shanghai Key Lab Bioenergy Crops, Coll Life Sci, Shanghai, Peoples R China.
7.[7]Fudan Univ, Dept Physiol & Pathophysiol, Basic Med Coll, Shanghai, Peoples R China.
8.[8]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Shanghai Key Lab Bioenergy Crops, Coll Life Sci, Shanghai, Peoples R China.
9.[9]Fudan Univ, Dept Physiol & Pathophysiol, Basic Med Coll, Shanghai, Peoples R China.
推荐引用方式
GB/T 7714
Hu, Li[1],Zhang, Shutian[2],Wen, Haoyu[3],et al. Melatonin decreases M1 polarization via attenuating mitochondrial oxidative damage depending on UCP2 pathway in prorenin-treated microglia[J]. PLOS ONE,2019,14:e0212138.
APA Hu, Li[1].,Zhang, Shutian[2].,Wen, Haoyu[3].,Liu, Tianfeng[4].,Cai, Jian[5].,...&Xia, Chunmei[9].(2019).Melatonin decreases M1 polarization via attenuating mitochondrial oxidative damage depending on UCP2 pathway in prorenin-treated microglia.PLOS ONE,14,e0212138.
MLA Hu, Li[1],et al."Melatonin decreases M1 polarization via attenuating mitochondrial oxidative damage depending on UCP2 pathway in prorenin-treated microglia".PLOS ONE 14(2019):e0212138.
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